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SEEDANCE 2.0 : The Most Insane AI Fight You’ve Ever Seen!

# Hollywood is cooked!

Seedance 2.0 just changed the AI video generation game forever. Forget everything you know about Sora or Veo. In this 16-minute cinematic masterclass, we push this AI model to its absolute limits to create the ultimate visual crossover. From hyper-realistic magic clashes inspired by the Marvel universe, to epic sci-fi battles worthy of Star Wars, and mind-blowing Pokémon realistic adaptations, the visual quality is simply unmatched.

Is Sora officially over? Watch this full showcase to see the insane capabilities, volumetric lighting, and face consistency that Seedance 2.0 can generate.

Don’t forget to LIKE and SUBSCRIBE to support the channel and never miss the latest AI tech news! Tell me in the comments which fight scene blew your mind the most. 👇

🇫🇷 FR : Seedance 2.0 vient de détruire la concurrence. Dans cette masterclass de 15 minutes, découvrez les combats générés par IA les plus épiques jamais créés. Des affrontements magiques intenses aux batailles spatiales, en passant par des créatures hyper-réalistes, la qualité cinématographique de Seedance 2.0 repousse toutes les limites. Pensez à vous abonner pour soutenir la chaîne!

Continue reading “SEEDANCE 2.0 : The Most Insane AI Fight You’ve Ever Seen!” | >

Abstract: A therapeutic target for terminating the long-lived autoimmune response!

Maureen A. Su find the epigenetic regulator UTX complexes with transcription factors TCF1 and STAT3 to promote pathogenicity of long-lived, stem-like progenitor T cells in models of type 1 diabetes (T1D)

1Department of Microbiology, Immunology, and Molecular Genetics, UCLA David Geffen School of Medicine, Los Angeles, California, USA.

2Department of Pediatrics and.

3Department of Genetics, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina, USA.

Complement Inhibition for Acute Neuromyelitis Optica Spectrum Disorder AttacksInsights From an International Case Series

These findings underscore the potential role of complement inhibition as a treatment option for acute NMOSD attacks, particularly in patients with insufficient response to standard therapies.

Background and Objectives.

Profile of a pimple

Sunny Y. Wong & team characterize gene expression changes that occur during acne pathogenesis and identify a therapeutic that reduces acne-like pustule formation in a mouse model of high fat diet-induced folliculitis:

The image shows prominent neutrophilic pustules from ear skin of the mouse model, stained for the neutrophil marker Ly6G (green) and KRT14 (red).

Address correspondence to: Sunny Y. Wong or Joseph S. Durgin, Department of Dermatology, University of Michigan, 1,500 E. Medical Center Dr., Ann Arbor, Michigan 48,109, USA. Email: sunnyw@umich.edu (SYW); durginj@med.umich.edu (JSD).

Norepinephrine acts through radial astrocytes in the developing optic tectum to enhance threat detection and escape behavior

Benfey et al. find that norepinephrine shifts the visual response selectivity of optic tectal neurons in the Xenopus tadpole to favor threatening loom stimuli over more neutral, randomly drifting dots. Mechanistically, norepinephrine induces radial astrocyte activation and glial release of ATP/adenosine, resulting in reduced excitatory neurotransmission and selectivity shift.

Japanese government panel endorses Sumitomo Pharma’s iPS-derived treatment for Parkinson’s

The expert council of the Japanese Ministry of Health has approved two regenerative medicine drugs based on induced pluripotent stem cells (iPSCs) — a cure for Parkinson’s disease and heart failure. If the health minister approves the experts’ decision, Japan will become the first country in the world to allow the commercial sale of such products — almost 20 years after the discovery of the technology itself.

Japan’s health ministry said on Thursday a panel of specialists had endorsed Sumitomo Pharma’s iPS cell-derived treatment for Parkinson’s disease, paving the way for the world’s first medical products based on the technology.

DOT1L provides transcriptional memory through PRC1.1 antagonism

Genetic off switch for cancer face_with_colon_three #cancer #cure

Neville, Ferguson et al. show that non-canonical Polycomb repressive complex 1.1-mediated gene silencing is antagonized by DOT1L and is required for the therapeutic efficacy of Menin and DOT1L inhibitors in mixed-lineage leukaemia.

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