For the first time, scientists have captured a stunning image of plasma’s “spaghetti-like” instability, revealing a phenomenon that could reshape our understanding of energy and fusion.
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Planarian worms can regenerate into a more youthful version of themselves
As you age you naturally lose neurons and muscle mass and experience a decline in fertility and wound healing ability. Previous research in animals has offered several potential techniques for turning back the biological clock in specific tissues, including exercise and calorie restriction. However, age reversal of blood cells or at whole organism level has so far been elusive.
HUGE Light Speed Computing Breakthrough
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CERN Is Secretly Collapsing Quantum Fields to Alter Local Gravity
Discover how CERN’s research into quantum fields could revolutionize our understanding of gravity! This deep dive explores the theoretical possibilities of manipulating quantum fields and their potential connection to gravitational forces. From Einstein’s predictions to cutting-edge experiments at the Large Hadron Collider, we examine what’s really happening at the frontier of physics research.
Learn how quantum gravity research could potentially transform:
Space travel and propulsion systems 🚀
Revolutionary energy production ⚡
Medical applications and treatments 🏥
Breakthroughs in Stopping Aging
Japanese researchers are making groundbreaking discoveries on the mechanisms of aging and working to apply them. As we age, senescent cells, or aged cells that have stopped dividing, accumulate, causing inflammation that can damage blood vessels and organs. Animal experiments have shown that removing these cells improves kidney function and reduces arteriosclerosis. They have led to the identification of a drug and development of a vaccine to eliminate the cells.
NAD+ Controls Circadian Reprogramming through PER2 Nuclear Translocation to Counter Aging
Disrupted sleep-wake and molecular circadian rhythms are a feature of aging associated with metabolic disease and reduced levels of NAD+, yet whether changes in nucleotide metabolism control circadian behavioral and genomic rhythms remains unknown. Here, we reveal that supplementation with the NAD + precursor nicotinamide riboside (NR) markedly reprograms metabolic and stress-response pathways that decline with aging through inhibition of the clock repressor PER2. NR enhances BMAL1 chromatin binding genome-wide through PER2 K680 deacetylation, which in turn primes PER2 phosphorylation within a domain that controls nuclear transport and stability and that is mutated in human advanced sleep phase syndrome.