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New artificial skin for bionic arm or AI robot | breakthrough photonic chip processes 2 billion images per second without memory device.


AI news includes new artificial skin to let AI robot, bionic arm or prosthetic limb feel with extreme touch sensitivity. New photonic chip allows AI to process and classify 2 billion images per second without needing storage device.

AI News Timestamps:
0:00 AI Robot Artificial Skin For Bionic Arm.
3:28 Photonic Chip Processes 2 Billion Images / Second.

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#AI #Robot #Bionic

Several years ago, a promising therapeutic using stem cell factor (SCF) emerged that could potentially treat a variety of ailments, such as ischemia, heart attack, stroke and radiation exposure. However, during clinical trials, numerous patients suffered severe allergic reactions and development of SCF-based therapeutics stopped.

A research team led by engineers at The University of Texas at Austin has developed a related therapeutic that they say avoids these major allergic reactions while maintaining its therapeutic activity. The keys to the discovery, published recently in Nature Communications, were the use of a similar, membrane-bound version of SCF delivered in engineered lipid nanocarriers.

“We envision this as something you can inject where you have lack of blood flow and it could induce to grow in that area,” said Aaron Baker, a professor in the Cockrell School of Engineering’s Department of Biomedical Engineering, and one of the leaders on the project.

In celebration of Pride Month, Inara, one of our moderators, is fundraising to pay for her transition surgery. I donated.

Gender dysphoria is the term used to describe a sense of discomfort or distress that a person may experience because of a mismatch between their gender identity and their sex assigned at birth. Methods of relieving gender dysphoria include transitioning socially (pronoun usage, name change, etc.) and physically (surgery).

I have medical insurance but it does not cover trans healthcare. Yes, we are still fighting for trans rights! I need allies willing to take action on my behalf.

To assess whether a compound holds promise for treating a disease, researchers usually begin by studying its use in animals. This allows us to see if the compound has a chance of curing the disease.

Animal models, however, rarely reproduce all aspects of a disease. The alternative is to represent the disease in cell cultures. While at first glance, Petri dishes look quite different from a person with a disease, the reality could be quite different when you look at them more closely.

Alzheimer’s has been cured more than 400 times in laboratories. How then can we still consider Alzheimer’s to be incurable? The reason is that it has only been cured in animals.

A new colorectal cancer drug has shocked researchers with how effective it is against the highly dangerous disease, after it virtually cured every member of a clinical trial.

Dostarlimab, a monoclonal antibody drug that is already approved to treat endometrial cancer in the UK, smashed expectations in a trial at Memorial Sloan Kettering Cancer Center in New York.

One year after the trial ended, each of the 18 participants’ cancer had gone into remission, with doctors unable to find signs of the cancer in their body.

Sascha Roth remembers the phone call came on a hectic Friday evening.

She was racing around her home in Washington, D.C., to pack for New York, where she was scheduled to undergo weeks of radiation therapy for rectal cancer. But the phone call from Memorial Sloan Kettering Cancer Center (MSK) medical oncologist Andrea Cercek changed everything, leaving Sascha “stunned and ecstatic — I was so happy.”

Dr. Cercek told Sascha, then 38, that her latest tests showed no evidence of cancer, after Sascha had undergone six months of treatment as the first patient in a clinical trial involving immunotherapy at MSK.


Rectal cancer patients saw their tumors disappear in a clinical trial involving immunotherapy at Memorial Sloan Kettering Cancer Center—without surgery, radiation, or chemotherapy.

New research has uncovered how genetic changes that accumulate slowly in blood stem cells throughout life are likely to be responsible for the dramatic change in blood production after the age of 70.

The study, by scientists at the Wellcome Sanger Institute, the Wellcome-MRC Cambridge Stem Cell Institute and collaborators, has been published in the journal Nature.

Longevity. Technology: Has our understanding of one of the mechanisms of aging taken a quantum leap? Molecular damage accumulates throughout our lives, gradually increasing year-on-year as we suffer telomere attrition, mutation, epigenetic change and oxidative and replicative stress. It’s a double whammy as our ability to repair this damage also declines as we age, but given the gradual nature of these processes, why, as the paper authors themselves put it, “Is there an abrupt increase in mortality after 70 years of age? [1].

A study published last week in Molecular Cell took a step towards that radical new concept for CRISPR. Led by Dr. Jennifer Doudna at the University of California Berkeley, who shared a Nobel Prize as a pioneer in the field, the study honed in on Cas9’s less famous and far more enigmatic cousin, Cas12c.

It’s the black sheep of the Cas family. Unlike other members, Cas12c completely lacks the ability to cut DNA. Instead, in bacteria cells, it binds onto invading viruses and protects vulnerable cells without shredding the virus’s DNA. The end result is a powerful antiviral defense system that doesn’t tax the host cell’s inner workings—yet makes it invincible to certain viral infections.

The study shows that chopping up viral DNA isn’t the only route for antiviral defense, at least in bacteria cells, the authors said. But more importantly, we’ve only begun scratching the surface of CRISPR gene editors.