The workshop discusses the recent issue Systems Neuroscience Through The Lens of Evolutionary Theory edited by Paul Cisek and Ben Hayden published by the Royal Society:
https://royalsocietypublishing.org/toc/rstb/2022/377/1844
Continue reading “Systems Neuroscience Through the Lens of Evolutionary Theory Workshop” »
Chinese scientists in a 10-year study found that people who adhered to the special six rules, the risk of dementia decreased by 90%. Even with a genetic predisposition to Alzheimer’s disease. The study involved 29 thousand people over 60 years of age, who were divided into three groups.
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Persons with autism spectrum conditions (ASC) often engage in video gaming, one of the most common leisure activity in this population. Escapism, aimed at the avoidance of negative experiences or self-development, is considered as one of the main gaming motivations. Furthermore, escapism is a self-regulatory strategy used while suffering from autistic burnout, consisting of exhaustion, reduced social skills, anhedonia, and withdrawal. The goal of the current study was to determine predictors of escapism in video gaming among adult gamers with ASC. It was hypothesized that two types of escapism – self-suppression and self-expansion – would differentiate gaming motivations, affective outcomes, anhedonia, and autistic burnout rates. A total of 189 persons participated in the study (Mage = 27.52, SDage = 7.25), including 105 females. The results obtained indicated that self-suppression escapism was predicted by introjected regulation, positive and negative affect, and hedonic tone (F = 8.760, p < .001), while self-expansion was predicted by identified and integrated gaming motivations, hedonic tone, and positive affect (F = 23.066, p < .001). PLS-SEM analysis revealed good fit of the model with autistic burnout predicting self-suppression escapism. These results acknowledge the two-dimensional approach to escapism and highlight potential risk factors of self-suppression, especially among persons presenting symptoms of autistic burnout. Future research and clinical application directions are outlined.
Some philosophers search for the mark of the cognitive: a set of individually necessary and jointly suΜcient conditions identifying all and only the instances of cognition. They claim the mark is necessary to answer diΜcult questions concerning the nature and distribution of cognition.
Here, I will argue that, as things stand, given the current landscape of cognitive science, we are not able to identify a mark of the cognitive. I proceed as follows. First, I clarify some factors motivating the search for the mark of the cognitive, thereby highlighting the desiderata the mark is supposed to satisfy. Then, I highlight a tension in the literature over the mark. Given the literature, it is not clear whether the search aims for a mark capturing the intuitive notion of cognition or a genuine scientiΞc kind.
Researchers have been working for many years to comprehend the relationship between brain structure, functional connectivity, and intelligence. A recent study provides the most comprehensive understanding to date of how different regions of the brain and neural networks contribute to a person’s problem-solving ability in a variety of contexts, a trait known as general intelligence.
The researchers recently published their findings in the journal Human Brain Mapping.
The research, led by Aron Barbey, a professor of psychology, bioengineering, and neuroscience at the University of Illinois Urbana-Champaign, and first author Evan Anderson, a researcher for Ball Aerospace and Technologies Corp. working at the Air Force Research Laboratory, employed the technique of “connectome-based predictive modeling” to evaluate five theories on how the brain leads to intelligence.
Scientists have developed a minuscule brain implant smaller than a human hair which allows people to control computers with their minds. The chip is developed by Precision Neuroscience and works along similar principles to those of Elon Musk’s Neuralink company, though the Precision chip sits on the brain rather than in the tissue.
Summary: Injecting the brain molecule into mouse models of multiple sclerosis increased the number of oligodendrocytes. The findings suggest fractalkine may help to slow the progression, or potentially halt multiple sclerosis.
Source: University of Alberta.
A University of Alberta researcher is one step closer to demonstrating the potential of a brain molecule called fractalkine to halt and even reverse the effects of multiple sclerosis and other neurodegenerative diseases.
Its impressive how adaptive the brain is to each situation. It can sense which portions of the brain need more blood flow depending on energy usage and makes the needed tiny adjustments.
Summary: Researchers identified a specific type of cell that sits on top of the brain’s smallest blood vessels that sense when their region of the brain is in need of energy.
Source: University of Maryland
Even a simple movement like pushing a button sends ripples of activity throughout networks of neurons spanning across the brain, new University of Oregon research shows.
The finding highlights just how complex the human brain is, challenging the simplified textbook picture of distinct brain areas dedicated to specific functions.
“It’s really well known that that the primary motor cortex controls movement output,” said Alex Rockhill, a graduate student in the lab of human physiology professor Nicki Swann. “But there’s a lot more to movement than this one brain area.”
𝐀 𝐝𝐫𝐮𝐠 𝐭𝐡𝐚𝐭 𝐢𝐧𝐜𝐫𝐞𝐚𝐬𝐞𝐬 𝐝𝐨𝐩𝐚𝐦𝐢𝐧𝐞 𝐜𝐚𝐧 𝐫𝐞𝐯𝐞𝐫𝐬𝐞 𝐭𝐡𝐞 𝐞𝐟𝐟𝐞𝐜𝐭𝐬 𝐨𝐟 𝐢𝐧𝐟𝐥𝐚𝐦𝐦𝐚𝐭𝐢𝐨𝐧 𝐨𝐧 𝐭𝐡𝐞 𝐛𝐫𝐚𝐢𝐧 𝐢𝐧 𝐝𝐞𝐩𝐫𝐞𝐬𝐬𝐢𝐨𝐧, 𝐄𝐦𝐨𝐫𝐲 𝐬𝐭𝐮𝐝𝐲 𝐬𝐡𝐨𝐰𝐬
𝘼𝙣 𝙀𝙢𝙤𝙧𝙮 𝙐𝙣𝙞𝙫𝙚𝙧𝙨𝙞𝙩𝙮 𝙨𝙩𝙪𝙙𝙮 𝙥𝙪𝙗𝙡𝙞𝙨𝙝𝙚𝙙 𝙞𝙣 𝙉𝙖𝙩𝙪𝙧𝙚’𝙨 𝙈𝙤𝙡𝙚𝙘𝙪𝙡𝙖𝙧 𝙋𝙨𝙮𝙘𝙝𝙞𝙖𝙩𝙧𝙮 𝙨𝙝𝙤𝙬𝙨 𝙡𝙚𝙫𝙤𝙙𝙤𝙥𝙖, 𝙖 𝙙𝙧𝙪𝙜 𝙩𝙝𝙖𝙩 𝙞𝙣𝙘𝙧𝙚𝙖𝙨𝙚𝙨 𝙙𝙤𝙥𝙖𝙢𝙞𝙣𝙚 𝙞𝙣 𝙩𝙝𝙚 𝙗𝙧𝙖𝙞𝙣, 𝙝𝙖𝙨 𝙥𝙤𝙩𝙚𝙣𝙩𝙞𝙖𝙡 𝙩𝙤 𝙧𝙚𝙫𝙚𝙧𝙨𝙚 𝙩𝙝𝙚 𝙚𝙛𝙛𝙚𝙘𝙩𝙨 𝙤𝙛 𝙞𝙣𝙛𝙡𝙖𝙢𝙢𝙖𝙩𝙞𝙤𝙣 𝙤𝙣 𝙗𝙧𝙖𝙞𝙣 𝙧𝙚𝙬𝙖𝙧𝙙 𝙘𝙞𝙧𝙘𝙪𝙞𝙩𝙧𝙮, 𝙪𝙡𝙩𝙞𝙢𝙖𝙩𝙚𝙡𝙮 𝙞𝙢𝙥𝙧𝙤𝙫𝙞𝙣𝙜 𝙨𝙮𝙢𝙥𝙩𝙤𝙣𝙨 𝙤𝙛 𝙙𝙚𝙥𝙧𝙚𝙨𝙨𝙞𝙤𝙣.
Numerous labs across the world have shown that inflammation causes reduced motivation and anhedonia, a core symptom of depression, by affecting the brain’s reward pathways.
