This looks quite interesting: MMR vaccines might be beneficial against #Covid19
PDF | Published epidemiological data suggests a correlation between patients who receive measles-rubella containing vaccines such as the commonly… | Find, read and cite all the research you need on ResearchGate.
Even before the pandemic, public health agencies around the world were struggling to counter increasingly sophisticated efforts to turn people against vaccines. With vaccination rates against measles and other infectious diseases falling in some locations, the World Health Organization (WHO) in 2019 listed “vaccine hesitancy” as one of 10 major global health threats.
To stop the pandemic, the world’s public health experts must win the coming “story war” over vaccine misinformation.
Well this is interesting:
A Henry Ford Health System study shows the controversial anti-malaria drug hydroxychloroquine helps lower the death rate of COVID-19 patients, the Detroit-based health system said Thursday.
Officials with the Michigan health system said the study found the drug “significantly” decreased the death rate of patients involved in the analysis.
The study analyzed 2,541 patients hospitalized among the system’s six hospitals between March 10 and May 2 and found 13% of those treated with hydroxychloroquine died while 26% of those who did not receive the drug died.
Continue reading “Hydroxychloroquine lowers COVID-19 death rate, Henry Ford Health study finds” »
What if your seat on the subway could fight off coronavirus? A French company has designed a new type of ‘decontaminating’ fabric, which is already equipping local metro trains.
Do the stimulus checks being sent to individuals to help in the coronavirus crisis count as a form of universal basic income? Andrew Yang thinks so. But, as WSJ’s Jason Bellini reports, others believe the intention behind UBI is misguided. Photo: Tom Brenner/Getty Images.
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Thank you my hero.
Elon Musk has joined the global tech crusade now underway to revolutionize vaccine manufacturing — now aimed at delivering billions of doses of a new mRNA vaccine to fight Covid-19. And he’s cutting right to the front.
In a late-night tweet Wednesday, the Tesla chief announced:
Vitaliy Karimov/Shutterstock
Tesla, the electric car company founded and run by Elon Musk, is building mobile molecular printers to assist Germany’s CureVac in manufacturing its experimental COVID-19 vaccine. Musk tweeted the information on Wednesday, July 1.
The “printers” are portable, automated messenger RNA (mRNA) production units, which Musk referred to as “RNA microfactories.”
Lurking just beneath the surface of just about every common nursery rhyme is a complex record of times long gone. For example, the “crooked man” who “laid a crooked sixpence upon a crooked style” was none other than the great 17th-century Scot General Sir Alexander Leslie. The crooked stile was the uneasy border between Scotland and England established by the controversial covenant he signed. Quite similarly, many enigmatic structures that permanently persist or otherwise transiently appear and resorb in the development of the nervous systems of many creatures also encode a rich evolutionary past.
One such functioning relic is Reissner’s fiber, a glycoprotein sheet secreted by the subcommissural organ (SCO) that inexorably treadmills down the central canal of the spinal cord. Although the SCO was one of the first structures of the mammalian brain to differentiate, in humans, it begins regressing around age three or four and typically becomes vestigial by adulthood. The main component of Reissner’s fiber is a giant 5000-amino-acid vertebrate molecule called SCO-spondin. This protein contains axonal pathfinding domains critical to development of the posterior commissure, a transhemispheric highway that bears axons controlling the pupillary light reflex.
The other product of the SCO is a thyroid-hormone-transporting protein called transthyretin. Much like all the organified metals fixed by life, iodine has a unique story to tell in the evolution of the body plan. Recently, an intriguing connection between Reissner’s fiber and development of the spine that houses it has been discovered in the model organism, zebrafish. These fish, as recently observed for the serotonergic control of neurogenesis, have proven to be an exemplary model for studying all things neural. In the latest issue of Current Biology, author Nathalie Jurisch-Yaksi reviews a remarkable confluence of ideas that establish an indisputable role for Reissner’s membrane building a straight and strong spine.
If there’s one myth that has persisted through the years without much evidence, it’s this: multiply your dog’s age by seven to calculate how old they are in “human years.” In other words, the old adage says, a four-year-old dog is similar in physiological age to a 28-year-old person.
But a new study by researchers at University of California San Diego School of Medicine throws that out the window. Instead, they created a formula that more accurately compares the ages of humans and dogs. The formula is based on the changing patterns of methyl groups in dog and human genomes — how many of these chemical tags and where they’re located — as they age. Since the two species don’t age at the same rate over their lifespans, it turns out it’s not a perfectly linear comparison, as the 1:7 years rule-of-thumb would suggest.
A team of researchers affiliated with several institutions in South Korea has found that stimulating production of a certain enzyme in roundworms can increase their lifespan. In their paper published in the journal Science Advances, the group describes their study of the protein VRK-1 and what they learned about its impact on the longevity of roundworms.
Prior research has shown that one way to increase longevity in some species is to use techniques that slow down mitochondrial respiration. In this new effort, the researchers were looking to better understand why slowing energy use in mitochondria has an impact on aging. As part of their effort, they looked at an energy sensor in mitochondria called adenosine 5’-monophosphate-activated protein kinase (AMPK), known to play a role in controlling how much energy is used in cells in roundworms. Prior research had suggested its level of activity is controlled by the protein VRK-1. To learn more about its impact on aging, the researchers genetically engineered two lines of roundworms to force them to produce more VRK-1 and two lines of roundworms to force them to produce less VRK-1. They then monitored the roundworms to see how long they lived.
The researchers found those roundworms expressing more than the normal amount of VRK-1 tended to live longer than average, while those expressing less than average amounts of VRK-1 had shorter lifespans. More specifically, control worms representing the normal lifespan of a roundworm lived on average 16.9 days. In their experiments, one of the lines expressing more VRK-1 lived on average 20.8 days, while the other lived on average 23.7 days. And one of the lines producing less VRK-1 lived on average just 12.7 days and the other just 15.9 days. The researchers suggest this finding indicates that VRK-1 has a direct impact on roundworm longevity.
